The Plastic Surgery Foundation
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Grants We Funded

Grant Applicants for 2020 requested more than $4.1 million. The PSF Study Section Subcommittees of Basic and Translational Research and Clinical Research Evaluated 111 applications on the following topics:

The PSF awarded Research Grants totaling more than $860,000 to support 24 plastic surgery research proposals.

The American Society of Plastic Surgeons/PSF leadership is committed to continuing to provide high levels of investigator-initiated research support to ensure that plastic surgeons have the needed research resources to be pioneers and innovators in advancing the practice of medicine.

Research Abstracts

Search The PSF database to have easy access to full-text grant abstracts from past PSF-funded research projects 2003 to present. All abstracts are the work of the Principal Investigators and were retrieved from their PSF grant applications. Several different filters may be applied to locate abstracts specific to a particular focus area or PSF funding mechanism.

Circulating Endothelial Progenitor Cells and Diabetic Wound Healing

Principal Investigator
Robert Allen MD

Year
2008

Institution
New York University Medical Center

Funding Mechanism
Basic Research Grant

Focus Area
Wounds/Scar

Abstract
Many factors contribute to the impairment of wound healing in diabetic patients. Diabetic wound healing may be impaired due to macro and micro angiopathy, decreased blood flow, decreased immune response, decreased availability of stem or progenitor cells involved in neovascularization and tissue regeneration, and decreased cellular activity due to glycosylation. The purpose of this study is to investigate the role of circulating endothelial progenitor cells in diabetic wound healing. With increased knowledge of the mechanisms resulting in impaired diabetic wound healing, we may be able to better treat these patients or even prevent the occurrence of chronic wounds all together. We hypothesize that impaired diabetic wound healing is in part a result of a decreased population of circulating endothelial progenitor cells (EPC) as well as an inability of these cells to migrate to ischemic tissues. We propose that improving these processes or circumventing them altogether will lead to improved diabetic wound healing.